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Concurrently, MAPK pathways (p38 and JNK) are also activated, increasing inflammatory responses via phosphorylation of transcription factors such as CREB and AP-1 [63]

Thus, considering dose response of APAP in future study designs will be crucial in improving the mechanistic understanding of the differences between spontaneous transplant-free survival versus transplant-assisted survival and ALF-related deaths observed in APAP overdose patients