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Description

This study demonstrates that the combination of spirochaetal and host genetics, in this case infection with an RST1 strain in patients with a TLR1 polymorphism (1805GG), leads to greater inflammation, more-severe early disease and a greater frequency of antibiotic-refractory Lyme arthritis

To define the synergy between GSTP1 and HPV-16 E7 and the role of their interaction in enhancing cell survival after UV exposure, we used siRNA to down-regulate GSTP1 protein levels