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These results led us to directly examine the benefits of NQO1 transgenesis (NQO1-Tg), and its contribution to improved insulin sensitivity and metabolic dysfunction by performing an integrated analysis of physiological data, histological changes, and omics profiles from serum and metabolically active tissues such as liver, skeletal muscle, and visceral fat in mice fed either standard diet (SD) or high-fat diet (HFD)

The approval is supported by three Phase III trials, including INTEGUMENT-1 and INTEGUMENT-2

This process is fundamentally dependent on the activation of the AhR and subsequent upregulation of the autophagy-associated gene ATG5 (138, 139)

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