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[DOI] [PMC free article] [PubMed] [Google Scholar] Yang SR, Valvo S, Yao H, Kode A, Rajendrasozhan S, Edirisinghe I, Caito S, Adenuga D, Henry R, Fromm G, Maggirwar S, Li JD, Bulger M, Rahman I

References de novo synthesis but not recycling process coordinates with glutamine catabolism to control redox homeostasis and directs murine T cell differentiation

Although the incidence is low, the mortality rate is extremely high

On the other hand, FoB rely on OXPHOS to a lower extent: Gclc -deficient FoB lose CI and CII functionality, but these cells increase glycolysis to replenish their ATP pool to the extent that they persist in Gclc fl/fl Mb1 Cre + hosts