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Mitochondria, as the core source and main target of ROS [449], their functional damage will further form a positive feedback loop of ROS generation - mitochondrial damage, causing energy metabolism disorders and inducing mitochondria to release pro-apoptotic factors such as cytochrome C, ultimately leading to neuronal apoptosis [450]

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Increases in the concentrations of long- and very-long-chain fatty acids, suggesting their impaired -oxidation, were reported in the serum of ASD children [13,14]