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doi: 10.1111/j.1753-4887.2008.00118.x 40 StamenkovicA.PierceG

In conclusion, our study uncovers a novel post-translational regulatory mechanism by which LDHB-K58 delactylation promotes gastric cancer metastasis through SLC7A11-mediated GSH synthesis and ferroptosis resistance

Notably, NSC-based interventions have also been investigated in the context of tumors arising from dysregulated NSCs programs, such as gliomas, primarily as vehicles for targeted drug delivery (Fawzy et al., 2025)