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In this manner, prenatal exposure to environmental toxicants or alterations in prenatal nutrients could predispose the mitochondria to be sensitive to post-natal physiological stressors [91], such as an infection or inflammatory event or other toxicant exposures

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Hence, it is noteworthy to thoroughly explore parasites and their derived molecules to potentially develop novel gene therapeutics that may help in managing cancer as a controllable disease

This will reduce the protection provided by the HPA axis, attenuating immune and inflammatory changes, and leaving ME/CFS patients more vulnerable to minor stressors (429)