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While our mechanistic data and mouse model results are compelling, a formal statistical causal inference or randomized controlled design would be required to definitively attribute hyperprogression to the immunotherapy regimen

M., Grieneisen, V

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Mononuclear MDSCs (M-MDSCs) have been shown to be implicated in resisting immune checkpoint inhibitors (anti-CTLA-4 and anti-PD-1) and are associated with receptor tyrosine kinase (KIT) signaling in CRC (100)